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Humic Acid Definition and Usage

Researchers believe that this is the missing link to optimal health and nutrition, which can change life for the better. Experts agree that this nutritional element should be used in conjunction with vitamins and minerals as a key dietary requirement. Since the discovery of this substance, pharmaceutical companies have rushed to have synthetic versions of the substance patented. However, it is naturally available in a much more potent form than the synthetic version, which could never be copied. humic acid is formed by Earth itself. The ancient deposits in the soil, is commonly known as humus. It is that organic part of the soil that stays behind after centuries of microbial decomposition of vegetation and animals. Coal and oil form as a result of further decomposition. Humic deposits are another strata, which at first was most unusual to geologists. The compound is dark in color and alkaline soluble. The substance acts as an acceptor or donor of electrons, depending on what is needed. Basically, it will bind to both negatively and positively charged electrons. This makes humic acid the most powerful scavenger of free radicals and a powerful natural anti-oxidant. Humic acid is a powerful immune booster. It also helps the body to manufacture glycoproteins which attach to killer T-cells. It acts as a modulator or a link between the cells in order to regulate the immune cells and preventing the cells from becoming unbalanced. T-cells play an important role in the immune response of the body. Cytotoxic Thymus cells destroy infected cellules. When activated, the T-cells secrete cytokines and proliferate. These cells are attacked by the HIV infection and when there is a decrease in CD4+ T cells, AIDS is the result. Regulatory T-cells, otherwise known as suppressor T-cells suppress the immune system to maintain a homeostasis. Autoimmune diseases result from this homeostasis of the immune system as immunocytes attack the healthy cells in the same body. This is often seen in victims of burn incidents and radiation sickness. The dead cells are attacked and this creates unwanted infections. However, humates cause the immune system to reduce infection by recognizing its own dead cellules. Several institutions are researching topical and internal humates to see how infections can be reduced in burn victims. Fulvic acid is a component of humate and is a very powerful chelator with unique abilities. Minerals that are life sustaining are placed in a phyto-state (chemical state) to make them readily absorbed by the organisms or cells. Toxic cells are chelated too and put in a chemical state that makes it harder to be absorbed by the cellules. It removes heavy metals including mercury and lead from the bloodstream. In soil, fulvic acid acts as a filter for toxic metals by grabbing and immobilizing it to prevent it from migrating or causing a chemical reaction. Crops grown in fulvic deficient soil can absorb toxins. The soil in which our food is currently grown is nutritionally depleted, which is why there is simply not enough nutrition in the food. This is why it is necessary to take humic acid supplements.

More on Cystic Fibrosis Part 4

Cystic fibrosis also affects the digestive system. In a healthy person, the pancreas produces chemicals (enzymes) which pass into the gut as food leaves the stomach. These enzymes break down the fat. If you have cystic fibrosis, the pancreas does not produce enzymes. Without these enzymes, the fat in food is not properly digested and it is difficult to gain weight. The feces contain an excess of fat and are oily and very smelly (BUPA, 2006). The presentation of unusual feces along with ongoing chest symptoms and failure to thrive in children are the three main factors which physicians will observe for when considering a diagnosis of cystic fibrosis.

The exocrine pancreas is the most profoundly affected gastrointestinal organ in cases of cystic fibrosis. Approximately 85-90% of cystic fibrosis patients suffer from pancreatic insufficiency from birth (Davis et al, 1996). Untreated pancreatic insufficiency can reduce fat absorption to as low as 40-50% and protein absorption is also impaired (Gow et al, 1981). The primary objective in the cystic fibrosis patient is to maintain normal growth into adulthood with the use of pancreatic replacement therapy and other nutritional support. Enzyme preparations are usually taken in capsule form and are available as a powder for young children. The drug is taken with every meal or snack to ensure that the fat and protein absorption is optimized.

Genetic Involvement
Cystic fibrosis is a genetically inherited disease caused by a faulty gene called the cystic fibrosis transmembrane conductance regulator – or CFTR gene. In order to inherit the disease a faulty gene must be passed from both the father and the mother. In cases where only one faulty gene is passed the individual will then be a ?carrier? and not have acquired the disease themselves.
When both parents are carriers, with each pregnancy there is a:

? 1 in 4 chance of having a child with cystic fibrosis
? 1 in 2 chance of having a child who is a carrier
? 1 in 4 chance of having an unaffected child

There are several different types of genetic mutation which are associated with different degrees of severity of the disease (BUPA, 2006).

Due to advances in medical science cystic fibrosis can be diagnosed whilst the baby is still in the uterus. The use of chorionic villus sampling (CVS) allows for the detection of the disease before the baby is even born. In cases where both parents are known carriers awareness of whether the unborn infant has the disease is often desirable. The finding of two mutations of the disease confirms that it is present however the finding of only one does not ultimately exclude it. Siblings of a cystic fibrosis patient are at equal risk of having the disease, and often is the case that following diagnosis of one child, parents request that all children be screened, this is usually offered as a standard service to most families anyway.

Research into Cystic Fibrosis
The Cystic Fibrosis Foundation established a Research Development Program Center for research in cystic fibrosis with a five-year, $2 million grant in 1997. It was renewed in 2002 and 2007. The primary goal of the Center is to focus the attention of new and established investigators on multidisciplinary approaches designed to improve the understanding and treatment of cystic fibrosis.

The Center?s research efforts focus on several areas relevant to the understanding and treatment of cystic fibrosis: basic studies of the function, protein interactions, trafficking and processing of the cystic fibrosis gene product, CFTR; understanding the infection-inflammation issues that compromise the function of CF airways; the development of new therapies and diagnostic approaches for treating cystic fibrosis, and participation of Center investigators in clinical studies.